Muaj zog^®H. pylori antibodody cov tshuaj ntsuam sai sai yog qhov pom kev nrawm rau Helicobacter rau Helicobacter rau Helicobpacter rau Helicobpacter rau Helicobpacter.

Cov txiaj ntsig
Nrawm thiab yooj yim
Cov ntshav ntiv tes tau siv tau.
Chav Chav Kub

Cov lus qhia
Rhiab 93.2%
Tshwj xeeb 97.2%
Qhov tseeb 95.5%
CE cim
Cov Khoom Siv Loj = 20 qhov kev ntsuam xyuas
Cov Ntaub Ntawv: Phau Ntawv / MSDS

Lub hauv paus rau phau ntawv
Gastritis thiab peptic mob yog nyob ntawm feem ntau cov tib neeg cov kab mob.Txij li qhov kev tshawb pom ntawm H. pylori (Warren & Marshall, 1983), ntau daim ntawv ceeb toomtau tawm tswv yim tias tus kab mob no yog ib qho ntawm cov loj ua rau ulcerCov Kab Mob (Anderson & Nielsen, 1983; kev tua tsiaj & Mohamed, 1995; Lambert LiAl, 1995). Txawm hais tias lub luag haujlwm ntawm H. pylori tseem tsis tau nkag siab meej,Kev cuam tshuam ntawm H. pylori tau cuam tshuam nrog kev tshem tawm ntawm cov kiav txhabKab mob. Tib neeg cov lus teb rau kev kis mob H. pylori muajtau pom (Varia & Holton, 1989; Evans et al, 1989). Kev kuaj pomntawm IGG Antibodies tshwj xeeb rau H. pylori tau pom tias yog qhov tseebTxoj kev rau kev kuaj pom H. pylori kis tau tus neeg mob. H. pylori
yuav colortize cov neeg asymptomatic. Kev kuaj mob serological yuav raug sivtxawm yog ib qho kev sib txuas lus rau endoscopy lossis raws li lwm txoj kev ntsuas hauvCov qauv mob.

Lub ntsiab lus
H. pylori antibododody ceev ceev sim ntaus ntawv (tag nrho cov ntshav / ntshav / ntshav / tshuaj tiv thaivIGM thiab IGG Antibodies tshwj xeeb rau Helicobacter pylori los ntawm kev pomKev txhais cov xim kev txhim kho ntawm txoj hlua sab hauv. H. pylori antigens yogimmobilized rau ntawm qhov kev sim ntawm daim nyias nyias. Thaum kuaj, hnoos qeevreacts nrog H. pylori antigen conjugated rau cov xim thiab prepoatedrau cov qauv pad ntawm qhov kev xeem. Qhov sib tov ces migrates los ntawmmembrane los ntawm capillary txiav txim, thiab cuam tshuam nrog reagents ntawm daim nyias nyias. YogMuaj cov tshuaj tiv thaiv txaus rau Helicobacter pylori hauv cov hnoos qeev, xim ximBand yuav tsim nyob rau ntawm thaj chaw kuaj ntawm daim nyias nyias. Lub xub ntiag ntawm cov xim noBand qhia txog qhov txiaj ntsig zoo, thaum nws tsis yog qhov tshwm sim tsis zoo. Tuscov tsos ntawm cov xim sib deev ntawm cov cheeb tsam tswj tau ua cov txheej txheemtswj, qhia tau tias cov ntim ntim kom zoo tau muab ntxiv thiabMembrane Wicking tau tshwm sim.

COV KEV CEEV FAJ
• Rau kev paub txog vitro kuaj xyuas siv nkaus xwb.
• Tsis txhob siv tom qab hnub tas sij hawm uas tau qhia ntawm pob. Tsis txhob sivQhov kev xeem yog tias cov ntawv nyiaj yog puas. Tsis txhob siv xeem xeem.
• Cov khoom siv no muaj cov khoom lag luam ntawm keeb kwm. Muaj ntawv pov thawj ntawm Phau Ntawv MaumoosKeeb kwm thiab / lossis huv lub xeev ntawm cov tsiaj tsis lees paub kiag liqhov tsis muaj cov neeg ua kom muaj transmissibenic. Nws yog yog li ntawd,pom zoo tias cov khoom no tau kho kom zoo li kev sib kis tau, thiabdaws los ntawm kev soj ntsuam ib txwm muaj kev nyab xeeb ceev (piv txwv li, tsis txhob ingest lossis nqus tau).
• Zam kev hla cov hnoos qeev los ntawm kev siv lub khob ntim khoom tshiab rau txhua qhov hnoos qeev tau.
• Nyeem tag nrho cov txheej txheem ua tib zoo ua ntej kuaj.
• Tsis txhob noj, haus lossis haus luam yeeb nyob rau txhua qhov chaw uas cov hnoos qeev thiab cov khoom siv tau raug daws.Kov txhua yam khoom uas yog tias lawv muaj cov neeg sawv cev sib kis. Saib xyuas tauKev ceev faj kev tawm tsam cov kev phom sij microbiological thoob plaws lubCov txheej txheem thiab ua raws cov txheej txheem txheem txheem rau cov khoom pov tseg kom raug.Hnav cov khaub ncaws tiv thaiv xws li kuaj cov tsho ntev, cov hnab looj tes ntawm hnab looj tes thiab lub qhov muagKev tiv thaiv thaum muaj cov qauv tsis tau.
• Cov ntaub ntawv me me muaj cov sodium Azide, uas tuaj yeem hnov ​​nrogua lossis tooj cov kav dej kom tsim cov hlau uas tawg tau yooj yim. Thaumpov tseg ntawm cov hnoos qeev kom tsis txhob ua cov qauv lossis cov qauv rho tawm, ib txwm uayaug nrog copious kom muaj cov dej kom tiv thaiv Azide Buildup.
• Tsis txhob muab kev sib haum xeeb lossis sib xyaw los ntawm ntau ntau.
• Cov av noo thiab kub tuaj yeem cuam tshuam cuam tshuam cuam tshuam rau cov txiaj ntsig.
• Siv cov ntaub ntawv xeem yuav tsum muab pov tseg raws li cov cai hauv zos.

Ntawv Sau Npe
1. Andersen LP, Nielsen H. Peptic Iluic: Ib qho kis tau mob li cas? Ann med. 1993Dec; 25 (6): 563-8.
2. Evans DJ JR, Evans Dg, Graham DY, Klein PD. Ib tug rhiab thiab tshwj xeebKev kuaj xyuas cov serologic rau kev nrhiav tau ntawm Campylobacter pylori muaj mob.Gastroenterology. 1989 Apr; 96 (4): 1004-8.
3. Kev yos hav zoov Rh, Mohamed Ah. Lub luag haujlwm tam sim no ntawm Helicobacter pylorieradication nyob rau hauv kev tshawb fawb. Scand j Gastroenaler. 1995; 208:47-52.
4. LAMBER JR, lin sk, aranda-Michel J. Helicobacter pylori. Scand jGastroenterol pab. 1995; 208: 33-46.
5. YTGAT GN, REWS EA. Lub luag haujlwm ntawm Campylobacter pylori hauvCov kab mob goltrodual. Tus "believer" 's taw tes saib.Gastroenterol CLAS BIOL. 1989; 13 (1 PT 1): 118b-121b.
6. Vaira d, Holton J. Ntshav Pleev Plumblogulin G Antibody QibCampylobacter pylori kev kuaj mob. Gastroenterology. 1989 Oct;97 (4): 1069-70.
7. Warren JR, Marshall B. Unidentified nkhaus Bacilli ntawm Paus Hauv Paus Apithelium hauvnquag ua rau mob plab zom mov. Lancet. 1983; 1: 1273-1275.

Ntawv muaj